By Ramana S. Moorthy MD
Starts with an in-depth review of immunemedicated eye ailment, summarizing easy immunologic techniques, ocular immune responses and particular issues in ocular immunology. Discusses the scientific method of uveitis and reports noninfectious (autoimmune) and infectious types of uveitis, with an elevated part on viral uveitis and new fabric on infectious and noninfectious scleritis. stronger detection of infectious brokers through immunologic and genetic tools and new biologic therapeutics are distinctive. additionally covers endophthalmitis, masquerade syndromes, problems of uveitis and ocular facets of AIDS. includes a variety of new colour pictures. significant revision 2011-2012
Read or Download 2011-2012 Basic and Clinical Science Course, Section 9: Intraocular Inflammation and Uveitis (Basic & Clinical Science Course) PDF
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Extra info for 2011-2012 Basic and Clinical Science Course, Section 9: Intraocular Inflammation and Uveitis (Basic & Clinical Science Course)
The basic immunoglobulin structure is composed of 4 covalently bonded glycoprotein chains that form a monomer of approximately 150,000- 180,000 daltons (Fig 2-6). Each antibody monomer contains 2 identical light chains, either kappa (K) or lambda (A), and 2 identical heavy chains from 1 of the 9 structurally distinct subclasses Antigen binding site Antigen binding site yNH NH, NHX' f-----,'<-- Hinge region Constant region eOOH eOOH Figure 2-6 Schematic represe ntation of an im munog lobulin molecule.
Cytotoxic T lymphocytes express CDB and serve as effector cells for killing tumors or virally infected host cells through release of cytotoxic cytokines or specialized pore-forming molecules. A third subset of effector lymphocytes. grouped as non -T. non-B lymphocytes. includes natural killer cells. lymphokine-activated cells. and ki ller cells. Antibodies. or immunoglobulins. are soluble antigen-specific effector molecules of adaptive immunity. After appropriate antigenic stimulation with T-Iymphocyte help.
IL-4 can also induce macrophage granulomas in response to parasite-derived antigens. 2-3 Ocular Inflammatory Diseases Thought to Require a Major Contribution of Th1-Mediated DH Effec10r Mechanisms Site Disease Presumed Antigen Conjunctiva Contact hypersensitivity to contact lens solutions Giant papillary co njun ctivitis Phlycten ulosis Chronic allog raft rejection Marginal infiltrates of blepharitis Disciform keratitis after vira l infection Acute anterior uveitis Sarcoidosis-associated uveitis Intermediate uveitis Sympathetic op hthalmia Vogt-Koyanagi- Harada syndrome Birdshot retinochoroidopathy Acute thyroid orbitopathy Giant cell arteritis Thim erosa l or other chemicals Cornea and sclera Anterior uvea Retina and choroid Orbit Unknown Bacteria l anti gens Histocompatibility antigens Bacterial antigens Viral antigens Uveal autoantigens.